Cancer

Carcinogenesis and Neoplasia ... a Companion Site.

site map

¤ Cancer ¤¤ adenoviruses ¤ amplification ¤ cancer staging ¤ carcinogenesis ¤ c-Fos ¤ c-Jun ¤ c-Myc § c-Rel ¤ c-Sis ¤ estrogen receptors ¤ gene amplification ¤ genetic predispositon ¤ HBV ¤ HIV ¤ HPV ¤ HTLV-I ¤ immune evasion ¤ irradiation ¤ malignant transformation ¤ metastasis ¤ mitogens ¤ mutagens ¤ MYC ¤ mutations ¤ neoplasia ¤ neoplastic mutations ¤ NF-κB ¤ non-mutagenic carcinogens ¤ oncogenes ¤ p53 ¤ proliferation ¤ proto-oncogenes ¤ radiation ¤ Ras ¤ Rb ¤ retroviral mechanisms of carcinogenesis ¤ retroviruses ¤ signaling molecules ¤ SRC genes ¤ staging ¤ T-antigens ¤ TP53 ¤ tumor antigens ¤ tumor suppressors ¤ tumorigenic viruses ¤ viral carcinogens ¤ v-Fos ¤ v-Sis ¤ v-Myc § v-Rel ¤


Glossary : A . B . C . D . E . F . G . H . I . J . K . L . M . N . O . P . Q . R . S . T . U . V . W . X . Y . Z .

Related topics on Companion Sites ◦◦A◦◦° adhesion ° adhesion molecules ° allosteric enzymes °Ape1 ° apoptosis ° AP endonuclease ° ◦◦B◦◦° base excision repair BER ° Bax ° Bcl-2 ° ◦◦C◦◦ ° cadherins ° cAMP-dependent protein kinase ° cap-dependent translation initiation ° capping ° CDK ° cell-cycle checkpoints ° cell-cycle control ° cellular adhesion molecules ° cellular differentiation ° cellular signal transduction ° cellular stress response ° chaperone ° chromatid, chromatin, chromosome ° cis versus trans-acting factors ° codon DNA RNA ° cyclin-dependent kinases ° cyclins ° cytokines ° cytokine receptors ° cytoskeleton ° C to U ° ◦◦D◦◦° DAGKs ° deletions ° diacyl glycerol kinases ° DNA damage, C to T, C to U ° DNA damage by ROS ° DNA ligases ° DNA mutation and alkylation ° DNA polymerases ° DNA repair ° DNase IV ° double strand breaks ° duplication ° ◦◦E◦◦° ° epigenetics ° ERKs ° evading immune system ° exon ° exonuclease 1 ° exosome °◦◦F◦◦ ° FAK ° FEN-1° Flap Endonuclease ° focal adhesion kinases (FAKs) ° ◦◦G◦◦ ° gene regulation ° genome ° gene regulation ° genetic template ° G-protein coupled receptors ° GPCRs ° GPCR families ° growth factor genes ° guanylate cyclases · guanyl cyclase °◦◦H◦◦ ° hOGG1 oxoG repair ° ◦◦I◦◦° ionizing radiation ° immune privilege ° immunoglobulins ° insertion ° integrins ° internal ribosomal entry ° intron ° inversion °◦◦L◦◦ ° LINEs ° long interspersed elements ° long terminal repeats ° LTRs °◦◦M◦◦° MAP kinase ° meiosis ° metabolic regulation ° mismatch repair ° mispairing ° mitochondrion ° mitogen activated protein kinases ° mitosis ° Msh2-Msh3 ° mutation ° MutS, MutL, and MutH ◦◦N ◦◦° non-receptor tyrosine kinases (PTKs) nonsense-mediated decay ° nonstop decay ° nucleolus ° nucleus ° ◦◦O◦◦ Oncogenes Proto-oncogenes ° open reading frame ° ORF ° oxidative stress and DNA damage ° oxoG repair ° ◦◦P◦◦° PCNA ° phosphatases ° phosphodiesterases ° phospolipases ° phosphotransfer-mediated signaling pathways ° phosphorylation ° polyadenylation ° promoters ° proteasome ° protein degradation ° protein tyrosine kinases °  Proto-oncogenes Oncogenes ° PTKs ◦◦R◦◦° ° Ras ° receptor proteins ° receptor tyrosine kinases ° recombinant DNA ° regulation ° regulatory proteins ° regulatory sequence ° replication ° replication factor C ° repressor ° response elements ° reproduction ° reverse transcriptase ° Rho-GTPase ° ribosomes ° ribosome shunting ° ribosomal structure °  RTKs ◦◦S◦◦ ° second messengers ° selectins ° Serine/Threonine Kinases ° signal transduction ° SINEs ° spindle ° spliceosomal-mediated RNA trans-splicing ° substitution ° ◦◦T◦◦° targetted genetic repair ° transcription ° transcription factors ° transcription initiation ° termination of transcription ° transposable elements ° transduction ° translation elongation ° translation initiation ° translation termination ° translocation ° transformation ° tRNA ° TG mismatch ° T U mismatch °◦◦U◦◦ ° ubiquitin ° ubiquitin ligase ° UvrD ° ◦◦X◦◦°XRCC1 ° ◦◦8◦◦° 8-oxoguanine glycosylase °

. . . proliferating since 10/06/06